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--- bacteria:t3e:xopaa [2025/01/29 22:56] – [Biological function] jfpothier
+++ bacteria:t3e:xopaa [2025/02/12 23:09] (current) – jfpothier
@@ Line 25: / Line 25: @@
 === Regulation ===
-RT-PCR expression analyses suggested that, although translocation of XopAA is dependent on the Hrp T3SS apparatus, //xopAA// expression does not depend on nutritional conditions that induce //hrp// gene expression in //Xee // (Morales //et al.//, 2005).
+RT-PCR expression analyses suggested that, although translocation of XopAA is dependent on the Hrp T3SS apparatus, //xopAA// expression does not depend on nutritional conditions that induce //hrp// gene expression in //Xee// (Morales //et al.//, 2005).
 === Phenotypes ===
-Transgenic rice plants expressing XopAA//<sub>Xoo</sub> // exhibited semi-dwarfism and a reduction in Brassinolide-dependent laminar inclination, characteristics of brassinosteroid (BR)-insensitive mutants caused by mutations of the BR receptor (Yamaguchi //et al.//, 2013).
+Transgenic rice plants expressing XopAA//<sub>Xoo</sub>// exhibited semi-dwarfism and a reduction in Brassinolide-dependent laminar inclination, characteristics of brassinosteroid (BR)-insensitive mutants caused by mutations of the BR receptor (Yamaguchi //et al.//, 2013).
-Rice plants inoculated with a T3SS-deficient //hrpX// mutant of //X. oryzae// pv. //oryzae// (//Xoo//) did not develop disease lesions. In contrast, a transgenic rice line overexpressing //xopAA// showed severe lesions when inoculated with the //Xoo// //hrpX// mutant and the bacterial population of the //hrpX// mutant was 100-fold higher in the transgenic plants than in wild-type plants. This observation led the authors conclude that XopAA<sub>//Xoo// </sub> inhibits resistance to //Xoo//, which was probably caused by suppression of host PTI (Yamaguchi //et al.//, 2013). However, a //xopAA// knock-out strain of //Xoo //did not exhibit any defect in virulence (Yamaguchi //et al.//, 2013).
+Rice plants inoculated with a T3SS-deficient //hrpX// mutant of //X. oryzae// pv. //oryzae// (//Xoo//) did not develop disease lesions. In contrast, a transgenic rice line overexpressing //xopAA// showed severe lesions when inoculated with the //Xoo// //hrpX// mutant and the bacterial population of the //hrpX// mutant was 100-fold higher in the transgenic plants than in wild-type plants. This observation led the authors conclude that XopAA<sub>//Xoo//</sub> inhibits resistance to //Xoo//, which was probably caused by suppression of host PTI (Yamaguchi //et al.//, 2013). However, a //xopAA// knock-out strain of //Xoo// did not exhibit any defect in virulence (Yamaguchi //et al.//, 2013).
 Expression of XopAA<sub>//Xoo//</sub> in //Arabidopsis// cells activated host immune responses, suggesting the presence of intracellular immune receptors that recognize XopAA (Yamaguchi //et al.//, 2013).

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