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--- bacteria:t3e:avrbs2 [2025/07/24 22:08] – jfpothier
+++ bacteria:t3e:avrbs2 [2025/07/24 22:10] (current) – jfpothier
@@ Line 86: / Line 86: @@
   * AvrBs2 has been demonstrated to be required for full virulence of //X. euvesicatoria// pv. //euvesicatoria// (aka //X. campestris// pv. //vesicatoria//), //X. oryzae// pv. //oryzicola//, //X. phaseoli// pv. //manihotis// (aka //X. axonopodis// pv. //manihotis//) (Zhao //et al//., 2011; Li //et al//., 2015; Mutka //et al.//, 2016; Medina //et al//., 2018).
   * Recognition of AvrBs2 by OsHRL makes rice more resistant against //X. oryzae// pv. //oryzicola// (Park //et al//., 2010).
-  * It was shown in pepper and tomato lines without //Bs2 //that mutations of catalytic residues in the glycerolphosphodiesterase did not interfere with the ability of the plant to recognize AvrBs2 through the cognate R gene //Bs2// and trigger disease resistance. This finding suggests that recognition of AvrBs2 is independent of its glycerolphosphodiesterase enzyme activity (Zhao //et al//., 2011).
+  * It was shown in pepper and tomato lines without //Bs2// that mutations of catalytic residues in the glycerolphosphodiesterase did not interfere with the ability of the plant to recognize AvrBs2 through the cognate R gene //Bs2// and trigger disease resistance. This finding suggests that recognition of AvrBs2 is independent of its glycerolphosphodiesterase enzyme activity (Zhao //et al//., 2011).
   * AvrBs2 contributes to //X. oryzae// pv. //oryzicola// virulence by suppressing PAMP-triggered defense responses in rice (Li //et al//., 2015).
   * AvrBs2 transiently expressed in //Arabidopsis// protoplasts suppressed flg22-induced NHO1 expression (Li //et al//., 2015).
@@ Line 110: / Line 110: @@
 Yes (//e.g.//, //X//. //arboricola//, //X//. //campestris//, //X//. //citri//, //X//. //euvesicatoria//, //X//. //fuscans//, //X//. //oryzae//, //X//. //phaseoli//).
-Field strains of //X. euvesicatoria// pv. //euvesicatoria// and //X//. //campestris// pv. //campestris// were found to accumulate mutations in the //avrBs2/////avrRxc1/3// gene in order to overcome //Bs2/////Rxc1/////Rxc3//-mediated resistance (Swords //et al.//, 1996; Gassmann //et al.//, 2000; Ignatov //et al.//, 2002). Yet, the global //Xcv// population was found to be extremely clonal, with very little genetic variation throughout the chromosome, including //avrBs2// and the plasmid-borne //avrBs1//, a finding that is consistent with recent evolution or population expansion of the species (Wichmann //et al.//, 2005).
+Field strains of //X//. //euvesicatoria// pv. //euvesicatoria// and //X//. //campestris// pv. //campestris// were found to accumulate mutations in the //avrBs2/////avrRxc1/3// gene in order to overcome //Bs2/////Rxc1/////Rxc3//-mediated resistance (Swords //et al.//, 1996; Gassmann //et al.//, 2000; Ignatov //et al.//, 2002). Yet, the global //Xcv// population was found to be extremely clonal, with very little genetic variation throughout the chromosome, including //avrBs2// and the plasmid-borne //avrBs1//, a finding that is consistent with recent evolution or population expansion of the species (Wichmann //et al.//, 2005).
 === In other plant pathogens/symbionts ===

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